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Zhou Xu
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Found 19 results
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Adaptation to DNA damage as a bet-hedging mechanism in a fluctuating environment. R Soc Open Sci. 8(8), pp.210460 (2021).
. Architecture and evolution of subtelomeres in the unicellular green alga Chlamydomonas reinhardtii. Nucleic Acids Res. pp.2021.01.29.428817 (2021).
. Telomere shortening causes distinct cell division regimes during replicative senescence in Saccharomyces cerevisiae. Cell Biosci. 11(1), pp.180 (2021).
. Maturation of telomere 3’-overhangs is linked to the replication stress response. bioRxiv. pp.2020.08.27.269621 (2020).
. Adaptation in replicative senescence: a risky business. Curr Genet. (2019).
. The many types of heterogeneity in replicative senescence. Yeast. (ja), (2019).
. Molecular characterization of Chlamydomonas reinhardtii telomeres and telomerase mutants. Life Sci Alliance. 2, (2019).
. Molecular characterization of Chlamydomonas reinhardtii telomeres and telomerase mutants. Life Science Alliance. 2, (2019).
. A subtelomeric region affects telomerase-negative replicative senescence in Saccharomyces cerevisiae. Scientific Reports. 9(1), pp.1845 (2019).
. Adaptation to DNA damage checkpoint in senescent telomerase-negative cells promotes genome instability. Genes Dev. 32(23-24), pp.1499-1513 (2018).
. Effects of initial telomere length distribution on senescence onset and heterogeneity. J Theor Biol. 413, pp.58-65 (2017).
. The asymmetry of telomere replication contributes to replicative senescence heterogeneity. Sci Rep. 5, pp.15326 (2015).
. The prion protein is critical for DNA repair and cell survival after genotoxic stress. Nucleic Acids Res. 43(2), pp.904-16 (2015).
. Two routes to senescence revealed by real-time analysis of telomerase-negative single lineages. Nat Commun. 6, pp.7680 (2015).
. The length of the shortest telomere as the major determinant of the onset of replicative senescence. Genetics. 194(4), pp.847-57 (2013).
. Dual conformation of H2H3 domain of prion protein in mammalian cells. J Biol Chem. 286(46), pp.40060-8 (2011).
. Mechanistic insights into cellular alteration of prion by poly-D-lysine: the role of H2H3 domain. FASEB J. 25(10), pp.3426-35 (2011).
. Prion fibrillization is mediated by a native structural element that comprises helices H2 and H3. J Biol Chem. 285(27), pp.21004-12 (2010).
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